トップ›研究者を探す›骨髄由来血管前駆細胞の動態解明とその機能制御法の開発

骨髄由来血管前駆細胞の動態解明とその機能制御法の開発

KAKEN 科学研究費助成事業データベース で見る
研究課題番号 KAKENHI-PROJECT-15390241
研究種目 基盤研究(B)
研究分野 生物系
医歯薬学
内科系臨床医学
循環器内科学
研究機関 東京大学
代表研究者 佐田 政隆
研究分担者 平田 恭信
研究分担者 斎浦 明夫
研究期間 開始年月日 2003/4/1
研究期間 終了年度 2004
研究ステータス 完了 (2004/4/1)
配分額（合計） 15,000,000 (直接経費 :15,000,000)
配分額（履歴） 2004年度：5,000,000 (直接経費 :5,000,000)
2003年度：10,000,000 (直接経費 :10,000,000)
キーワード 幹細胞
動脈硬化
分化
平滑筋細胞
内皮細胞
動員
増殖因子
血管
stem cells
atherosclerosis
differentiation
smooth muscle cell
endothelial cells
mobilization
growth factor
artery

研究成果

[雑誌論文] Vascular regeneration and remodeling by circulating progenitor cells

Sata, M, Nagai, R 2005

[雑誌論文] Potent inhibitory effect of sirolimus on circulating vascular progenitor cells.

Fukuda, D., Sata, M., Tanaka, K., Nagai, R. 2005

[雑誌論文] The angiotensin II type 1 receptor blocker valsartan attenuates graft vasculopathy.

Yamamoto, T., Sata, M., Fukuda, D., Takamoto, S. 2005

[図書] Cardiovascular Regeneration Therapies Using Tissue Engineering Approaches.(Mori, H., Matsuda, H. eds.)

Sata, M, Nagai, R 2005

[図書] Cardiovascular Regeneration Therapies Using Tissue Engineering Approaches. (Mori, H., Matsuda, H. eds.)

Sata, M, Nagai, R 2005

[雑誌論文] Potent inhibitory effect of sirolimus on circulating vascular progenitor cells.

Fukuda, D., Sata, M., Tanaka, K., Nagai, R. 2005

[雑誌論文] Statins augment collateral growth in response to ischemia but they do not promote cancer and atherosclerosis.

Sata, M., Nishimatsu, H., Osuga, J.I., Tanaka, K. 2004

[雑誌論文] Origin of neointimal cells in autologous vein graft.

Sata M., Nagai, R. 2004

[雑誌論文] Anti-monocyte chemoattractant protein-1 gene therapy attenuates graft vasculopathy.

Saiura, A., Sata, M., Hiasa, K., Kitamoto, S., Washida, M., Egashira, K., Nagai, R., Makuuchi, M. 2004

[雑誌論文] Inflammation, angiogenesis, and endothelial progenitor cells : How do EPCs find their place ?

Sata, M., Nagai, R 2004

[雑誌論文] Diverse contribution of bone marrow cells to neointimal hyperplasia after mechanical vascular injuries.

Tanaka K., Sata M., Hirata Y., Nagai R. 2003

[雑誌論文] Molecular strategies to treat vascular diseases ; Circulating vascular progenitor cell as a potential target for prophylactic treatment of atherosclerosis.

Sata, M. 2003

[雑誌論文] Little evidence for cell fusion between recipient and donor-derived cells.

Saiura A, Sata M, Washida M, Sugawara Y, Hirata Y, Nagai R, Makuuchi M. 2003

[雑誌論文] Molecular strategies to treat vascular diseases ; Circulating vascular progenitor cell as a potential target for prophylactic treatment of atherosclerosis.

Sata M. 2003

[雑誌論文] Absence of p53 leads to accelerated neointimal hyperplasia after vascular injury.

Sata, M., Tanaka, K., Ishizaka N., Hirata, Y., Nagai R. 2003

[雑誌論文] Circulating vascular progenitor cells contribute to vascular repair, remodeling and esion formation.

Sata, M. 2003

[雑誌論文] Circulating vascular progenitor cells contribute to vascular repair, remodeling and lesion formation.

Sata M. 2003

[雑誌論文] Diverse contribution of bone marrow cells to neointimal hyperplasia after mechanical vascular injuries.

Tanaka K., Sata M., Hirata Y., Nagai R. 2003

[雑誌論文] Absence of p53 leads to accelerated neointimal hyperplasia after vascular injury.

Sata, M., Tanaka, K., Ishizaka N., Hirata, Y., Nagai R. 2003

[雑誌論文] Little evidence for cell fusion between recipient and donor-derived cells.

Saiura A, Sata M, Washida M, Sugawara Y, Hirata Y, Nagai R, Makuuchi M. 2003